The high dependency unit formed a nine-member medical board for Bajandar on Tuesday to proceed with the surgeries to remove the wood-like warts
A 28-year-old man from Khulna known as the "Tree Man," for bark-like warts on his hands and feet, is set to undergo a second series of surgeries from Saturday to remove the growths.
The man, Abul Bajandar, who was diagnosed with epidermodysplasia verruciformis – a rare genetic condition commonly known as “tree man syndrome”, had undergone 25 operations from January 30, 2016 to May 26, 2018. The warts began to grow when he was 10.
Dr Samanta Lal Sen, coordinator of Sheikh Hasina National Burn and Plastic Surgery Institute at the Dhaka Medical College Hospital (DMCH), on Wednesday told the Dhaka Tribune that they are ready to start the second phase of operations.
“Bajandar’s warts have returned, with the lesions on his hands growing back to their previous shapes,” Dr Sen said.
The high dependency unit formed a nine-member medical board for Bajandar on Tuesday to proceed with the surgeries to remove the wood-like warts.
Prof Md Abul Kalam, project director of the institute, is leading the board, of which Dr Sen is a member.
On May 26, 2018, Bajandar fled the hospital accusing the doctors of negligence and misconduct, which the institute denied.
He returned to DMCH on Sunday, when his physical condition began to deteriorate again.
“It would have been a bit easier for us (to deal with the case) if he had returned to DMCH responding to our calls at least four months ago,” Dr Sen said.
“Now, Bajandar will need at least eight other operations,” he added.
According to Dr Sen, such type of genetic condition does not have a cure and needs regular treatment.
Bajandar is among less than half a dozen people worldwide afflicted with the rare genetic condition.
The cause of the condition is an inactivating PH mutation in either the Epidermodysplasia verruciformis (EVER) 1 or EVER2 genes, which are located adjacent to one another on chromosome 17. These genes play a role in regulating the distribution of zinc in the cell nuclei. Zinc is a necessary cofactor for many viral proteins, and the activity of EVER1-EVER2 complex appears to restrict the access of viral proteins to cellular zinc stores, limiting their growth.
Other genes have also rarely been associated with this condition. These include the ras homolog gene family member H.